Figure 5
From: Regulations of the key mediators in inflammation and atherosclerosis by Aspirin in human macrophages
ASA inhibition of NFκ B-p65 protein nuclear translocation. THP-1 macrophages were treated with 0, 600, 1200 μM ASA, nuclear proteins were extracted and analysed by Western blot (A). Signal intensities in the blots were also measured by Quantity One software and transformed to the bar graph (B). The decrease at600, 1200 μM ASA was statistically significant ((P < 0.05). Results are expressed as mean ±S.D, of three experiments with duplicated samples. * P < 0.05,compared to control.