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Table 4 Endpoint plasma inflammation- and coagulation-related markers (n = 21)

From: Fish-oil esters of plant sterols differ from vegetable-oil sterol esters in triglycerides lowering, carotenoid bioavailability and impact on plasminogen activator inhibitor-1 (PAI-1) concentrations in hypercholesterolemic subjects

 

PS-SO2

PS-OO

PS-FO

P value3

Inflammation

    

   TNF-α (ng/L)

1.03 ± 0.191

1.03 ± 0.21

1.06 ± 0.24

0.9046

   IL-6 (ng/L)

1.77 ± 0.22

1.91 ± 0.22

1.50 ± 0.17

0.1501

   CRP (mg/L)

1.42 ± 0.48

1.48 ± 0.49

1.71 ± 0.63

0.8656

   PSA4 (μg/L)

0.92 ± 0.10

0.84 ± 0.08

0.89 ± 0.09

0.6102

Coagulation

    

   PAI-1 (μg/L)

40.3 ± 11.5a

26.6 ± 5.5ab

20.5 ± 2.6b

0.0297

   Fibrinogen (g/L)

3.41 ± 0.15

3.42 ± 0.11

3.25 ± 0.11

0.2217

  1. 1 Values are means ± SE's.
  2. 2 CRP: C-reactive protein; IL-6: Interleukin-6; PAI-1: Plasminogen Activator Inhibitor 1; PSA: prostate specific antigen; PS-SO: plant sterols esterified with sunflower oil fatty acids; PS-OO: plant sterols esterified with olive oil fatty acids; PS-FO: plant sterols esterified with fish oil fatty acids; TNF-α: Tumor necrosis factor – α
  3. 3 P values obtained by repeated-measures ANOVA.
  4. a, bValues not sharing a common superscript letter are significantly different at P < 0.05.
  5. 4 Chronic inflammation was suggested to play a role in the pathogenesis and progression of benign prostatic hyperplasia [54], which is associated with elevated PSA levels; n = 10
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Lipids in Health and Disease

ISSN: 1476-511X