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Fig. 1 | Lipids in Health and Disease

Fig. 1

From: Resveratrol intake during pregnancy and lactation re-programs adiposity and ameliorates leptin resistance in male progeny induced by maternal high-fat/high sucrose plus postnatal high-fat/high sucrose diets via fat metabolism regulation

Fig. 1

Body weight (BW) and adiposity of male progeny at four-months-old. (a) The BW and (b) total and individual adipose tissue depots weight relative the control group. Mesenteric (M), retroperitoneal (R), epididymal (E), and subcutaneous (S) fats were collected to measure adipose tissue weight; (c) The average adipocyte counts per microscopic field. The individual effects of maternal high-fat/high-sucrose (HFHS) diet (Hit 1) and postnatal HFHS diet (Hit 2) and their interaction (Hit 1 × Hit 2) were estimated with a two-way ANOVA. (Abbreviations: CC, maternal/postnatal control diet; HC, maternal HFHS/postnatal control diet; CH, maternal control/postnatal HFHS diet; HH, maternal/postnatal HFHS diet; and HRH, maternal HFHS diet plus maternal resveratrol/postnatal HFHS diet; *compared with CC, P < 0.05; #compared with HC, P < 0.05; §compared with CH, P < 0.05 by a Mann-Whitney U test). The Mann-Whitney U test was used to evaluate the therapeutic effect of resveratrol; †P < 0.05. (n = 10–14)

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Lipids in Health and Disease

ISSN: 1476-511X